Kawm paub ntau ntxiv txog Blood Cell Analyzers (Blood Routine)
1. Dab tsi yog qhov ntsuas ntshav ntsuas ntshav?
Ntshav cell analyzer, tseem hu ua ntshav cell analyzer,ntshav cell analyzer, ntshav celltxee, thiab lwm yam., yog ib qho ntawm cov cuab yeej siv dav hauv kev kuaj mob hauv tsev kho mob. Cov ntshav-cov ntaub ntawv ntsig txog xws li concentration, hematocrit, thiab lwm yam.
2. Cov khoom dab tsi tuaj yeem kuaj xyuas cov qe ntshav dawb? Qhov tseem ceeb ntawm kev kuaj ntshav niaj hnub yog txhawm rau pom cov cim ntxov ntawm ntau cov kab mob hauv lub cev, kuaj xyuas seb puas muaj ntshav ntshav, seb puas muaj kab mob ntshav, thiab cuam tshuam txog kev ua haujlwm hematopoietic ntawm cov pob txha pob txha. Cov tshuaj ntsuam xyuas ua lub luag haujlwm tseem ceeb hauv kev sim, yog li cas cov khoom kuaj ntshav kuaj ntshav muaj?
Cov qe ntshav dawb suav (WBC) Peripheral ntshav leukocytes los ntawm hematopoietic qia hlwb hauv cov pob txha pob txha. Leukocytes suav nrog granulocytes, lymphocytes thiab monocytes. Ntawm lawv, granulocytes tau muab faib ua neutrophils, neutrophils hlwb, eosinophils thiab basophils. Kev suav cov qe ntshav dawb yog qhov ntsuas ntawm tag nrho cov naj npawb ntawm ntau hom qe ntshav dawb hauv cov ntshav.
Tus nqi siv rau cov neeg laus: (4-10) 109 / L. Cov menyuam yaus: (5-12) 109 / L. Cov Me Nyuam: (15-20) 109 / L.
1. Leukocytosis
(1) Physiological: Feem ntau pom nyob rau hauv premenstrual, cev xeeb tub, yug me nyuam, lactating cov poj niam, strenuous ce, excitement, haus dej haus, tom qab noj mov, thiab lwm yam. Neonatal thiab me nyuam mos yog siab tshaj cov laus.
(2) Pathology feem ntau pom nyob rau hauv ntau yam kab mob, kev puas tsuaj ntawm cov ntaub so ntswg loj los yog necrosis, leukemia, malignant qog, uremia, ntshav qab zib ketoacidosis, thiab mob hnyav ntawm cov tshuaj lom neeg xws li organophosphorus tshuaj tua kab thiab hypnotics. Cov tshuaj siv tshuaj kuj tuaj yeem ua rau cov qe ntshav dawb.
2. Leukopenia
(1) Cov kab mob feem ntau pom muaj mob khaub thuas, aplastic anemia, leukemia, thiab lwm yam.
(2) Sulfonamides, antipyretic analgesics, qee cov tshuaj tua kab mob, tshuaj tua kab mob, tshuaj tua kab mob, thiab lwm yam yuav tsum tau siv rau kev siv tshuaj.
(3) Cov kab mob tshwj xeeb xws li gram- kab mob tsis zoo (tus kab mob typhoid fever, paratyphoid fever), Mycobacterium tuberculosis infection, viral infection (rubella, hepatitis), parasitic infection (malaria), thiab lwm yam.
(4) Kev cuam tshuam ntawm lwm yam hluav taws xob, tshuaj (benzene thiab nws cov derivatives), thiab lwm yam. Muaj ntau yam cuam tshuam rau cov ntshav dawb suav, thiab yog tias tsim nyog, kev txiav txim siab yuav tsum tau ua ke nrog cov ntsuas xws li cov ntshav dawb suav thiab cov ntshav dawb. cell morphology.
【neutrophils】
1. neutropenia
(1) Acute infection or purulent infection, including local infection (abscess, furuncle, tonsillitis, appendicitis, otitis media, etc.); systemic infection (pneumonia, erysipelas, sepsis, scarlet fever, diphtheria, acute rheumatic fever). In mild infection, the percentage of white blood cells and neutrophils can be increased; in moderate infection, the count can be > 10.0 × 109/L; in severe infection, the count can be >20.0 109 / L, nrog pom tseeb hloov pauv.
(2) Poisoning uremia, ntshav qab zib ketoacidosis, acidosis, thaum ntxov mercury lom, lead lom, los yog hypnotics, organophosphorus lom.
(3) Los ntshav thiab lwm yam kab mob mob hnyav, mob hemolysis, postoperative, malignant qog, myeloid leukemia, cov ntaub so ntswg hnyav, myocardial infarction thiab vascular embolism, thiab lwm yam.
(4) Physiological see "leukocytosis".
2. neutropenia
(1) Cov kab mob typhoid, paratyphoid, malaria, brucellosis, qee yam kab mob viral (xws li kab mob siab B, qhua pias, mob npaws), kab mob ntshav, anaphylactic shock, aplastic anemia, siab cachexia, neutropenia lossis tsis muaj cov tsos mob, hypersplenism, kab mob autoimmune, thiab lwm yam. (2) Kev puas tsuaj rau cov hlau hnyav hnyav lossis cov organic lom lom, hluav taws xob puas, thiab lwm yam.
(3) Cov tshuaj Antineoplastic tshuaj, benzodiazepine sedatives, sulfonylurea insulin secretagogues, tshuaj tiv thaiv kab mob, tshuaj tua kab mob, tshuaj tiv thaiv kab mob, tshuaj tiv thaiv kab mob, qee yam tsis yog -steroidal anti- tshuaj tiv thaiv, thiab lwm yam.
【Lymphocytes】
1. lymphocytosis
(1) Lub sij hawm sib kis ntawm cov kab mob xws li hnoos hawb pob, kis kab mob mononucleosis, kab mob lymphocytosis, tuberculosis, kab mob qhua pias, qhua pias, rubella, mumps, kis kab mob siab, tuberculosis thiab lwm yam kab mob sib kis.
(2) Cov kab mob hematological xws li mob qog noj ntshav thiab mob qog noj ntshav thiab mob qog nqaij hlav leukemic lymphosarcoma tuaj yeem ua rau muaj qhov nce ntxiv hauv cov lymphocyte suav; aplastic anemia thiab agranulocytosis kuj tuaj yeem ua rau cov txheeb ze nce hauv feem pua ntawm lymphocyte. Tsis tas li ntawd, nws tseem tuaj yeem pom nyob rau lub sijhawm tsis lees txais tom qab hloov lub raum.
2. Cov theem mob ntawm lymphopenia kis kab mob, kab mob hluav taws xob, kab mob hauv cellular immunodeficiency, ntev - siv cov tshuaj hormones adrenal cortex lossis raug hluav taws xob, thiab lwm yam. Tsis tas li ntawd, thaum cov neutrophils nce vim ntau yam, lymphocytes tuaj yeem ua tau. raug txo kom tsawg. Cov qe ntshav liab suav (RBC) Cov qe ntshav liab yog cov khoom tsim muaj ntau tshaj plaws hauv cov ntshav. Raws li tus neeg nqa khoom ua pa, lawv tuaj yeem thauj cov pa roj carbon dioxide thaum nqa thiab tso pa oxygen mus rau ntau cov ntaub so ntswg ntawm lub cev, thiab tswj xyuas cov kua qaub - puag ncig thiab tiv thaiv kab mob. Cov ntshav liab suav yog ib qho ntawm cov cim tseem ceeb rau kev kuaj mob ntshav qab zib.
Tus nqi siv txiv neej: (4.0-5.5)1012/L. Poj niam: (3.5-5.0) 1012/L. Cov menyuam yug tshiab: (6.0-7.0) 1012 / L.
1. polycythemia
(1) Cov txheeb ze nce yog pom hauv ntuav hnyav, raws plab, tso zis ntau dhau, poob siab, tawm hws, thiab kub hnyiab. Vim muaj dej ntau ntau, cov ntshav plasma txo qis thiab cov ntshav yog concentrated, yog li ntawd cov concentration ntawm ntau yam hauv cov ntshav nce raws li. ib ntus tshwm sim.
(2) Absolute increase is seen in: ① Physiological increase, such as hypoxia and high altitude life, fetuses, newborns, strenuous exercise or physical labor, accelerated release of red blood cells from bone marrow, etc.; ② Pathological compensatory and secondary increases, Often secondary to patients with chronic cor pulmonale, emphysema, mountain sickness and tumors (renal cancer, adrenal tumor); ③ polycythemia vera, chronic bone marrow hyperfunction of unknown cause, the red blood cell count can reach (7.0-12.0) 1012 /L.
2. txo cov qe ntshav liab
Kev tsis txaus ntawm cov tshuaj hematopoietic Ua los ntawm kev noj zaub mov tsis txaus lossis malabsorption, xws li kab mob plab hnyuv, haus dej cawv, ib nrab eclipse, thiab lwm yam, ua rau tsis txaus cov tshuaj hematopoietic xws li hlau, folic acid, vitamins, lossis protein, tooj liab,
